Research identifies how time heals all wounds

Wound healing requires interactions between cells resident at the damaged site and infiltrating immune cells. Endothelial progenitor cells (EPCs) come from the bone marrow and are key to the production of new vessels. Here, Toshikazu Kondo and colleagues demonstrate that the chemokine CCL5 helps to direct the recruitment of EPCs to sites of wounding by acting on the chemokine receptor CCR5. These findings identify the CCR5/CCL5 axis as a potential target to promote healing. Eurekalert!